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Even for drugs with a different target, such as the experimental AD agents posiphen and phenserine that lower the rate of synthesis of amyloid-β precursor protein and thereby reduces the generation of the AD toxic species amyloid-β peptide [242], the molecular mechanism via it is achieved (translational regulation mediated via an iron response element) within the 5’-untranslated region of amyloid-β precursor protein mRNA [242, 243] is present within the 5’-untranslated region of α-synuclein [244, 245] and thereby providing a basis for the assessment of these agents in PD [246, 247]
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Volume:10mg – 10 vial lab pack (Save 10%)
Sodium Ascorbate – 150mg
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