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doi: 10.3389/fmicb.2025.1619101
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Quantity:8 x Masks
Paradoxically, CHOP deletion in lipopolysaccharide-induced AKI aggravates inflammation and tissue injury—a finding likely explained by long-term CHOP deficiency disrupting autophagy, supporting the idea that early-phase UPR signaling can be cytoprotective (150, 151)
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