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In the case of ischemia-reperfusion, the XO-associated abnormal ctROS synthesis leads to oxidative stress-mediated F-actin depolymerization, which induces mitochondrial dysfunction and fission, and apoptosis of the cardiac microvascular endothelial cells [84,85]
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[DOI] [PubMed] [Google Scholar] 6.Ruiz-Ortega M., Rayego-Mateos S., Lamas S., Ortiz A., Rodrigues-Diez R.R
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